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Research shows it enhances fibroblast migration to injury sites through the FAK-paxillin pathway, improves collagen fiber organization and cross-linking, promotes angiogenesis to improve nutrient delivery, and reduces inflammatory mediators while preserving beneficial healing signals

Point mutations and NNK site saturation libraries of PtGSTF8 were constructed by overlap extension PCR as previously reported 49 using primers T7 fw, T7 term and: PtGSTF8 (V12X): V12C13_rv and V12X_fw, PtGSTF8 (C13X): V12C13_rv and C13X_fw, PtGSTF8 (C13S): V12C13_rv and C13S_fw, PtGSTF8 (N108X): N108_rv, N108X_fw, PtGSTF8 (F112X): F112_rv, F112X_fw (Supplementary Table 4) using pANT35 as template