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Metformin, via the AMPK/ACC pathway, reshapes lipid utilization and reduces ACSL4 and 4-HNE positivity, thereby improving chondrocyte resistance to ferroptosis ( in vitro models, inhibiting cartilage chondroptosis and improving OA ( Excessive lipid metabolic reprogramming in OA significantly raises ferroptosis risk, so targeting lipid synthesis, transport, and degradation represents a promising upstream approach to reducing ferroptotic damage

(2024) Evaluating the impact of testosterone replacement therapy on carotid atherosclerosis: a systematic review and meta-analysis Sigalos JT, Pastuszak AW

A significant inter-group difference in glycemic levels was noted: participants in the KD group exhibited stable glucose levels (60140 mg/dl), whereas the standard diet group showed post-meal glucose spikes for 2 hours, with baseline levels of 80100 mg/dl rising to 150180 mg/dl during meals

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